No title
Background Insulin resistance is common in adults with obstructive sleep apnoea (OSA), yet the extent to which longitudinal metabolic improvement is driven by OSA phenotype, continuous positive airway pressure (CPAP) treatment, or changes in body weight remains unclear. We examined 1-year changes in insulin resistance across OSA and non-OSA subgroups in the non-diabetic RICCADSA cohort. Methods In
